Course Content
Module 1 — Principles of Hormone Therapy in Oncology
Mechanism and Dosing Overview Aromatase Inhibitors (AIs): block the enzyme aromatase (CYP19A1), which converts androgens to estrogens in adipose and peripheral tissues. Examples: Anastrozole 1 mg orally daily, Letrozole 2.5 mg orally daily, Exemestane 25 mg orally daily. These are the preferred agents in postmenopausal women with estrogen receptor–positive breast cancer. Selective Estrogen Receptor Modulators (SERMs): such as Tamoxifen 20 mg orally daily — bind the estrogen receptor and act as antagonists in breast tissue but partial agonists in bone and endometrium. Selective Estrogen Receptor Degraders (SERDs): such as Fulvestrant 500 mg intramuscularly on days 0, 14, 28, then every 28 days thereafter — promote estrogen receptor degradation. CYP17 Inhibitors: Abiraterone acetate 1000 mg orally daily on an empty stomach plus Prednisone 5 mg orally twice daily — block the CYP17A1 enzyme, suppressing androgen synthesis in adrenal glands and tumors. Gonadotropin-Releasing Hormone (GnRH) Agonists and Antagonists: Leuprolide 7.5 mg subcutaneously every 4 weeks or depot every 3–6 months; Degarelix 240 mg subcutaneously loading dose, then 80 mg every 4 weeks; Relugolix 120 mg orally daily — suppress testosterone or estrogen production by downregulating or directly blocking pituitary gonadotropin release.
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Hormone Therapy for Cancer Patients: Mechanisms, Indications, and Clinical Practice
  • Androgen Deprivation Therapy (ADT): suppression/blockade of testosterone production/action.

  • Aromatase Inhibitor (AI): blocks aromatase (CYP19A1) → ↓ estrogen (anastrozole/letrozole/exemestane).

  • Selective Estrogen Receptor Modulator (SERM): tamoxifen—ER antagonist in breast, partial agonist in bone/endometrium.

  • Selective Estrogen Receptor Degrader (SERD): fulvestrant—binds ER and triggers degradation.

  • CYP17A1: androgen-synthesis enzyme targeted by abiraterone.

  • Gonadotropin-Releasing Hormone (GnRH): hypothalamic hormone controlling LH/FSH (agonists/antagonists suppress gonadal steroids).

  • DiHydroEpiAndrosterone (DHEA): androgen precursor supplement; avoid during endocrine therapy.

  • International Prostate Symptom Score (IPSS): urinary symptom scale, 0–35.

  • Intrauterine Device (IUD), Copper: non-hormonal contraception of choice during endocrine therapy.

  • Osteonecrosis of the Jaw (ONJ): serious antiresorptive complication—avoid implants/extractions on therapy.

  • RECIST v1.1: imaging response criteria.

  • Dual-Energy X-ray Absorptiometry (DEXA): bone mineral density scan.

  • EORTC QLQ-C30, SF-36, FACT-P: validated QoL instruments (oncology general, general health, prostate-specific).

  • ESR1/AR-V7: mutations/variants associated with endocrine resistance.